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Mechanistically, BPC-157 activates VEGFR2 via the PI3K-Akt-eNOS axis to promote angiogenesis, stimulates ERK1/2 signalling to enhance fibroblast proliferation and migration, and modulates the NO system to regulate vascular tone

and studies investigating the role of glutamatergic agonists to combat NMDAR hypofunction and reduce symptoms [7]

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N -acetylaspartylglutamate (NAAG) and N -acetylaspartate (NAA) in patients with schizophrenia